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neurosciencestuff:

Insomnia may cause dysfunction in emotional brain circuitry
A new study provides neurobiological evidence for dysfunction in the neural circuitry underlying emotion regulation in people with insomnia, which may have implications for the risk relationship between insomnia and depression.
“Insomnia has been consistently identified as a risk factor for depression,” said lead author Peter Franzen, PhD, an assistant professor of psychiatry at the University of Pittsburgh School of Medicine. “Alterations in the brain circuitry underlying emotion regulation may be involved in the pathway for depression, and these results suggest a mechanistic role for sleep disturbance in the development of psychiatric disorders.”
The study involved 14 individuals with chronic primary insomnia without other primary psychiatric disorders, as well as 30 good sleepers who served as a control group. Participants underwent an fMRI scan during an emotion regulation task in which they were shown negative or neutral pictures. They were asked to passively view the images or to decrease their emotional responses using cognitive reappraisal, a voluntary emotion regulation strategy in which you interpret the meaning depicted in the picture in order to feel less negative.
Results show that in the primary insomnia group, amygdala activity was significantly higher during reappraisal than during passive viewing.  Located in the temporal lobe of the brain, the amygdala plays an important role in emotional processing and regulation.
In analysis between groups, amygdala activity during reappraisal trials was significantly greater in the primary insomnia group compared with good sleepers. The two groups did not significantly differ when passively viewing negative pictures.
“Previous studies have demonstrated that successful emotion regulation using reappraisal decreases amygdala response in healthy individuals, yet we were surprised that activity was even higher during reappraisal of, versus passive viewing of, pictures with negative emotional content in this sample of individuals with primary insomnia,” said Franzen.
The research abstract was published recently in an online supplement of the journal SLEEP, and Franzen will present the findings Wednesday, June 5, in Baltimore, Md., at SLEEP 2013, the 27th annual meeting of the Associated Professional Sleep Societies LLC.
The American Academy of Sleep Medicine reports that about 10 to 15 percent of adults have an insomnia disorder with distress or daytime impairment. According to the National Institute of Mental Health, 6.7 percent of the U.S. adult population suffers from major depressive disorder. Both insomnia and depression are more common in women than in men.

neurosciencestuff:

Insomnia may cause dysfunction in emotional brain circuitry

A new study provides neurobiological evidence for dysfunction in the neural circuitry underlying emotion regulation in people with insomnia, which may have implications for the risk relationship between insomnia and depression.

“Insomnia has been consistently identified as a risk factor for depression,” said lead author Peter Franzen, PhD, an assistant professor of psychiatry at the University of Pittsburgh School of Medicine. “Alterations in the brain circuitry underlying emotion regulation may be involved in the pathway for depression, and these results suggest a mechanistic role for sleep disturbance in the development of psychiatric disorders.”

The study involved 14 individuals with chronic primary insomnia without other primary psychiatric disorders, as well as 30 good sleepers who served as a control group. Participants underwent an fMRI scan during an emotion regulation task in which they were shown negative or neutral pictures. They were asked to passively view the images or to decrease their emotional responses using cognitive reappraisal, a voluntary emotion regulation strategy in which you interpret the meaning depicted in the picture in order to feel less negative.

Results show that in the primary insomnia group, amygdala activity was significantly higher during reappraisal than during passive viewing.  Located in the temporal lobe of the brain, the amygdala plays an important role in emotional processing and regulation.

In analysis between groups, amygdala activity during reappraisal trials was significantly greater in the primary insomnia group compared with good sleepers. The two groups did not significantly differ when passively viewing negative pictures.

“Previous studies have demonstrated that successful emotion regulation using reappraisal decreases amygdala response in healthy individuals, yet we were surprised that activity was even higher during reappraisal of, versus passive viewing of, pictures with negative emotional content in this sample of individuals with primary insomnia,” said Franzen.

The research abstract was published recently in an online supplement of the journal SLEEP, and Franzen will present the findings Wednesday, June 5, in Baltimore, Md., at SLEEP 2013, the 27th annual meeting of the Associated Professional Sleep Societies LLC.

The American Academy of Sleep Medicine reports that about 10 to 15 percent of adults have an insomnia disorder with distress or daytime impairment. According to the National Institute of Mental Health, 6.7 percent of the U.S. adult population suffers from major depressive disorder. Both insomnia and depression are more common in women than in men.

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neurosciencestuff:

Insomnia may cause dysfunction in emotional brain circuitry
A new study provides neurobiological evidence for dysfunction in the neural circuitry underlying emotion regulation in people with insomnia, which may have implications for the risk relationship between insomnia and depression.
“Insomnia has been consistently identified as a risk factor for depression,” said lead author Peter Franzen, PhD, an assistant professor of psychiatry at the University of Pittsburgh School of Medicine. “Alterations in the brain circuitry underlying emotion regulation may be involved in the pathway for depression, and these results suggest a mechanistic role for sleep disturbance in the development of psychiatric disorders.”
The study involved 14 individuals with chronic primary insomnia without other primary psychiatric disorders, as well as 30 good sleepers who served as a control group. Participants underwent an fMRI scan during an emotion regulation task in which they were shown negative or neutral pictures. They were asked to passively view the images or to decrease their emotional responses using cognitive reappraisal, a voluntary emotion regulation strategy in which you interpret the meaning depicted in the picture in order to feel less negative.
Results show that in the primary insomnia group, amygdala activity was significantly higher during reappraisal than during passive viewing.  Located in the temporal lobe of the brain, the amygdala plays an important role in emotional processing and regulation.
In analysis between groups, amygdala activity during reappraisal trials was significantly greater in the primary insomnia group compared with good sleepers. The two groups did not significantly differ when passively viewing negative pictures.
“Previous studies have demonstrated that successful emotion regulation using reappraisal decreases amygdala response in healthy individuals, yet we were surprised that activity was even higher during reappraisal of, versus passive viewing of, pictures with negative emotional content in this sample of individuals with primary insomnia,” said Franzen.
The research abstract was published recently in an online supplement of the journal SLEEP, and Franzen will present the findings Wednesday, June 5, in Baltimore, Md., at SLEEP 2013, the 27th annual meeting of the Associated Professional Sleep Societies LLC.
The American Academy of Sleep Medicine reports that about 10 to 15 percent of adults have an insomnia disorder with distress or daytime impairment. According to the National Institute of Mental Health, 6.7 percent of the U.S. adult population suffers from major depressive disorder. Both insomnia and depression are more common in women than in men.

neurosciencestuff:

Insomnia may cause dysfunction in emotional brain circuitry

A new study provides neurobiological evidence for dysfunction in the neural circuitry underlying emotion regulation in people with insomnia, which may have implications for the risk relationship between insomnia and depression.

“Insomnia has been consistently identified as a risk factor for depression,” said lead author Peter Franzen, PhD, an assistant professor of psychiatry at the University of Pittsburgh School of Medicine. “Alterations in the brain circuitry underlying emotion regulation may be involved in the pathway for depression, and these results suggest a mechanistic role for sleep disturbance in the development of psychiatric disorders.”

The study involved 14 individuals with chronic primary insomnia without other primary psychiatric disorders, as well as 30 good sleepers who served as a control group. Participants underwent an fMRI scan during an emotion regulation task in which they were shown negative or neutral pictures. They were asked to passively view the images or to decrease their emotional responses using cognitive reappraisal, a voluntary emotion regulation strategy in which you interpret the meaning depicted in the picture in order to feel less negative.

Results show that in the primary insomnia group, amygdala activity was significantly higher during reappraisal than during passive viewing.  Located in the temporal lobe of the brain, the amygdala plays an important role in emotional processing and regulation.

In analysis between groups, amygdala activity during reappraisal trials was significantly greater in the primary insomnia group compared with good sleepers. The two groups did not significantly differ when passively viewing negative pictures.

“Previous studies have demonstrated that successful emotion regulation using reappraisal decreases amygdala response in healthy individuals, yet we were surprised that activity was even higher during reappraisal of, versus passive viewing of, pictures with negative emotional content in this sample of individuals with primary insomnia,” said Franzen.

The research abstract was published recently in an online supplement of the journal SLEEP, and Franzen will present the findings Wednesday, June 5, in Baltimore, Md., at SLEEP 2013, the 27th annual meeting of the Associated Professional Sleep Societies LLC.

The American Academy of Sleep Medicine reports that about 10 to 15 percent of adults have an insomnia disorder with distress or daytime impairment. According to the National Institute of Mental Health, 6.7 percent of the U.S. adult population suffers from major depressive disorder. Both insomnia and depression are more common in women than in men.

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fy-ra:

Ocean.
San Francisco, CA.

fy-ra:

Ocean.

San Francisco, CA.

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gtokio:

巨体とは裏腹に、頭部のどこかかわいらしい感じが素材の質感とギャップを生み、不思議な気配で満たします。 造形の大きさも相まって、刹那的なほどに無軌道に連なる鋼線の集積は更なる密度で異様なほどのカオスを立ちのぼらせ、把握不能な混沌となって迫ります。鋼線部分はむしろ幾何学的な風合いも備えているのですが、塊となったときの蠢くような異様な有機的な感触がアバンギャルドさを鮮烈に提示してきているように感じられ、その濃厚な気配にぐんぐんと呑み込まれていきます。
review:「matter」稲葉友宏、宮田聡志《5/21》: ex-chamber museum
this sculpture is made by tomohiro inaba at CASHI

gtokio:

巨体とは裏腹に、頭部のどこかかわいらしい感じが素材の質感とギャップを生み、不思議な気配で満たします。
造形の大きさも相まって、刹那的なほどに無軌道に連なる鋼線の集積は更なる密度で異様なほどのカオスを立ちのぼらせ、把握不能な混沌となって迫ります。鋼線部分はむしろ幾何学的な風合いも備えているのですが、塊となったときの蠢くような異様な有機的な感触がアバンギャルドさを鮮烈に提示してきているように感じられ、その濃厚な気配にぐんぐんと呑み込まれていきます。

review:「matter」稲葉友宏、宮田聡志《5/21》: ex-chamber museum

this sculpture is made by tomohiro inaba at CASHI

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I
inothernews:

A woman, identified as Cub Scout troop leader Ingrid Loyau-Kennett, confronts one of two suspects who moments before viciously beheaded a British soldier on a street in London in what government officials called a “terror-related” incident. The attackers had claimed responsibility for the murder, saying on camera “The only reason we have done this is because Muslims are dying every day. This British soldier is an eye for an eye a tooth for tooth.” Loyau-Kennett, who wasn’t hurt, is also captured on video telling one of the men “Right now it is only you versus many people. You are going to lose.  What would you like to do?”  Both suspects were later killed shot and wounded by police.    (Photo: @sibiillamlaw / Twitter via The New York Daily News)

inothernews:

A woman, identified as Cub Scout troop leader Ingrid Loyau-Kennett, confronts one of two suspects who moments before viciously beheaded a British soldier on a street in London in what government officials called a “terror-related” incident. The attackers had claimed responsibility for the murder, saying on camera “The only reason we have done this is because Muslims are dying every day. This British soldier is an eye for an eye a tooth for tooth.” Loyau-Kennett, who wasn’t hurt, is also captured on video telling one of the men “Right now it is only you versus many people. You are going to lose.  What would you like to do?”  Both suspects were later killed shot and wounded by police.    (Photo: @sibiillamlaw / Twitter via The New York Daily News)

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thescienceofreality:


Three Possible Futures for the Universe via Chandra X-ray Observatory (Credit: NASA/CXC/M.Weiss)“This illustration shows three possible futures for the Universe, depending on the behavior of dark energy, by showing how the scale of the Universe may change with time. If dark energy is constant, as the new Chandra results suggest, the expansion should continue accelerating forever. If dark energy increases, the acceleration may happen so quickly that galaxies, stars, and eventually atoms will be torn apart, in the so-called Big Rip. Dark energy may also lead to a recollapse of the Universe, in the Big Crunch. The illustration also shows the early decelerating expansion of the Universe, followed by the accelerating phase that started about 6 billion years ago.”

Related Link here on dark energy.

thescienceofreality:

Three Possible Futures for the Universe via Chandra X-ray Observatory (Credit: NASA/CXC/M.Weiss)

“This illustration shows three possible futures for the Universe, depending on the behavior of dark energy, by showing how the scale of the Universe may change with time. If dark energy is constant, as the new Chandra results suggest, the expansion should continue accelerating forever. If dark energy increases, the acceleration may happen so quickly that galaxies, stars, and eventually atoms will be torn apart, in the so-called Big Rip. Dark energy may also lead to a recollapse of the Universe, in the Big Crunch. The illustration also shows the early decelerating expansion of the Universe, followed by the accelerating phase that started about 6 billion years ago.”